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Assess Neurologic Symptoms, Chronicity, Volume Status, and ODS Risk
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Prevent Osmotic Demyelination and Hypertonic Saline Toxicity
High ODS risk includes sodium ≤105 mmol/L, alcohol use disorder, malnutrition, advanced liver disease, and hypokalemia. Target an initial 4–6 mmol/L rise. Limit correction to 8 mmol/L per 24 hours when ODS risk is high; at standard risk, avoid more than 10–12 mmol/L in 24 hours or 18 mmol/L in 48 hours. Hypertonic saline may cause volume overload, phlebitis, hypernatremia, and rapid overcorrection.
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Know Urea and Tolvaptan Toxicities
Urea may cause nausea, diarrhea, dysgeusia, and increased BUN; titrate within approximately 15–60 g/day to the sodium response. Tolvaptan may cause thirst, polyuria, hypernatremia, rapid correction, hypotension, and liver injury. Initiate in hospital, avoid strong CYP3A inhibitors, anuria, hypovolemia, inability to drink, and underlying liver disease, and limit use to 30 days. Major guidelines disagree about routine vaptan use; reserve it for selected resistant SIADH with specialist oversight. Avoid demeclocycline and lithium.
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Know Loop Diuretic and Glucocorticoid Toxicities
For clinically important congestion, a loop-naive adult may receive furosemide 20–40 mg IV; use a higher dose based on prior loop exposure. Monitor urine output, blood pressure, K, Mg, and creatinine because hypovolemia, AKI, hypokalemia, and hypomagnesemia may occur. Hydrocortisone can cause hyperglycemia, fluid retention, delirium, and infection risk; give glucocorticoid before thyroid hormone when adrenal insufficiency is possible.
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Avoid Isotonic Saline as Routine SIADH Treatment
When urine remains concentrated, isotonic saline may be excreted while retained water worsens hyponatremia. Do not routinely add salt tablets plus a loop diuretic; efficacy is limited and AKI or hypokalemia may occur.